[1] Zhang L, Li J, Wang B, et al. Signaling via dopamine D1 and D3 receptors oppositely regulates cocaine-induced structural remodeling of dendrites and spines
[J]. Neurosignals,2012,20(1):15-34.

[2] Li J, Zhang L, Gu JJ, et al. Cocaine-induced dendritic remodeling occurs in both D1 and D2 dopamine receptor-expressing neurons in nucleus accumbens
[J]. Neuroscience Letter, 2012,517(2): 118-122.

[3] Kiyoshi E, Silaja Y, Suhail K, et al. Basal ganglia dopamine loss due to defect in purine recycling
[J]. Neurobiol Dis, 2007,26(2):396-407.

[4] Ivan M, Lekha MR, Rosalinda C, et al. A Golgi study of neuronal architecture in a genetic mouse model for Lesch–Nyhan disease
[J]. Neurobiol Dis, 2005, 20(2):479 -490.

[5] Fenghua C, Torsten M, Gregers W. Repeated electrocon vulsive seizures increase the total umber of synapses in adult male rat hippocampus
[J]. Eur Neuropsychopharmacol, 2009,19(5):329-38.

[6] Maud MM, David JR, Gloria M. Increased Synapses in the Medial Prefrontal Cortex Are Associated With Repeated Amphetamine Administration
[J]. Synapse,2009 63(2):126-35.

[7]  吴洪娟, 张圣明,庄宝祥.超薄切片制备关键环节的处理
[J]. 解剖学杂志，2011, 34(1):133-134.

[8] Alcantara AA, Lim HY, Floyd CE, Cocaine- and morphine-induced synaptic plasticity in the nucleus accumbens
[J]. Synapse,2011,65(4):309-320.

[9] Akira I, Jody A, Ivan L, et al. Unbiased stereological quantication of neurons in the human spiral ganglion
[J]. Neuroscience Letters,2001,304 (1-2):93-96.